An Urgent Update to the Global Demyelination Epidemic Hypothesis – Sudden Deaths/Long COVID explained?

The WHO website itself shows that COVID Vaccination can induce MS/AI against Myelin

May 24, 2023

Following on with the hypotheses in one of my posts a few weeks back. Much better elaboration found here in this article on the specific mechanism by which the crazy sets in. Many scientists have noted the convergence found often found with myelination of the brain and psychopathy that only developed at a much older age ... so there is a pretty solid inference to draw here with the vaccines. This is an excellent stack, by the way. I recommend many articles here. -

Texas Arcane

This is an update to my post from yesterday. I saw that Long COVID and many of the Sudden Deaths occurring can be explained by demyelination. As I have continued to research this, I discovered that, indeed, COVID vaccination can induce MS. Or, to be more precise, an autoimmune attack against Myelin.

We report here two cases of multiple sclerosis (MS) with clinical and new radiological signs beginning in close temporal relation to spike (S) protein mRNA-based vaccinations.
Conclusion(s) Detailed studies of both peripheral blood- and CSF derived CD4+ T cells show that the onset of MS in these two cases is very likely caused by CD4+ T cell clones that cross-recognize SARS-CoV-2 S protein-derived peptides and peptides derived from myelin proteins, which have previously been implicated in MS.

Covid-19 vaccination can induce multiple sclerosis via cross-reactive CD4+ T cells recognizing SARS-CoV-2 spike protein and myelin peptides
https://pesquisa.bvsalud.org/global-literature-on-novel-coronavirus-2019-ncov/resource/pt/covidwho-2138820

Why this concerns me so much is that the Spike is almost certainly VERY attracted to the Brainstem.

While the possible causes of long-COVID include long-term tissue damage, viral persistence, and chronic inflammation, the review proposes, perhaps for the first time, that persistent brainstem dysfunction may also be involved. This hypothesis can be split into two parts. The first is the brainstem tropism and damage in COVID-19. As the brainstem has a relatively high expression of ACE2 receptor compared with other brain regions, SARS-CoV-2 may exhibit tropism therein.

Persistent Brainstem Dysfunction in Long-COVID: A Hypothesis
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7874499/

The most important piece of this puzzle is that we now have the mechanism for the observed Sudden Deaths in relation to the Brainstem Demyelination hypothesis.

In this case report, we present a patient with active relapsing-remitting MS who died unexpectedly due to the sudden onset of cardiac arrythmias. Post-mortem examination showed the presence of active demyelinating lesions in the medulla oblongata.

Sudden cardiac death in multiple sclerosis caused by active demyelination of the medulla oblongata
https://pubmed.ncbi.nlm.nih.gov/21586485/

We must endeavor to determine if this is, indeed, the case. We need histology of the Brainstem on all Sudden Cardiac Deaths in those under 60.

Vault-Co Communications

An Urgent Update to the Global Demyelination Epidemic Hypothesis – Sudden Deaths/Long COVID explained?

The WHO website itself shows that COVID Vaccination can induce MS/AI against Myelin